Publications

Wolfe DM, Padilla-Lopez S, Vitiello SP, and Pearce DA.  pH-dependent localization of Btn1p in the yeast model of Batten disease.  Disease Models and Mechanisms 4(1):120-5, 2011.  PMCID: PMC3008966.

Vitiello SP, Benedict, JW, Padilla-Lopez S, and Pearce DA.  Interaction between Sdo1p and Btn1p in the Saccharomyces cerevisiae model for Batten disease.  Human Molecular Genetics 1;19(5):931-42, 2010.  PMCID: PMC2816617.

Vitiello SP, Wolfe, D, and Pearce, DA. Absence of Btn1p in the yeast model for juvenile Batten disease may cause arginine to become toxic to yeast cells. Human Molecular Genetics 1;16(9):1007-16, 2007.  PMID: 17341489.

Phillips S, de Voer G, Taschner PEM, Korey C, Codlin S, Mole SE, and Pearce DA. Characterizing pathogenic processes in Batten disease: use of small eukaryotic model systems.  Biochimica Biophysica Acta 1762(10):906-19, 2006. PMID: 17049819.

Phillips S, Benedict, JW, Weimer JM, and Pearce DA. CLN3, the Protein Associated with Batten Disease: Structure, Function, and Localization.  Journal of Neuroscience Research 79:573-583, 2005.  PMID: 15657902.

Fang F, Phillips S, and Butler JS. Rat1p and Rai1p function with the nuclear exosome in the processing and degradation of rRNA precursors.  RNA 11(10):1571-8, 2005.  PMCID: PMC1370841.

Phillips S and Butler JS. Contribution of domain structure to the RNA 3’ end processing and degradation functions of the nuclear exosome subunit Rrp6p.  RNA 9:1098-1107, 2003.  PMCID: PMC1370474.