Publications


Moy, SS, Nonneman, RJ, Ghashghaei, HT, Weimer, JM, Yokota, Y, Lee, D, Lai, C, Threadgill, DW, and Anton, ES (2009) Deficient NRG1-ERBB Signaling Alters Social Approach: Relevance to Genetic Mouse Models of Schizophrenia. Journal of Neurodevelopmental Disorders, in press.

Weimer, JM, Yokota, Y, Stanco, A, Stumpo, DJ, Blackshear, P, and Anton, ES (2009) MARCKS modulates radial progenitor placement, proliferation, and organization in the developing cerebral cortex. Development. 136(17):2965-75.

Weimer, JM.*, Benedict, JW*, Amanda L Getty, AL, Pontikis, CC, Ming J Lim, MJ, Cooper, JD, Pearce, DA (2009) Cerebellar defects in a mouse model of juvenile neuronal ceroid lipofuscinosis. Brain Research, 1266:93-107. *Authors contributed equally to this work.

Weimer, JM, Stanco A, Cheng JG, Vargo AC, Voora S, Anton ES. (2008) A BAC transgenic mouse model to analyze the function of astroglial SPARCL1 (SC1) in the central nervous system. Glia. 56(9):935-41.

Ghashghaei, HT, Weimer, JM, Schmid, RS, Yokota, Y, Papko, B, Kudlow, JE, and Anton, ES (2007) Regaining of radial glail identity in the mature cerebral cortex: Reinduction of ErbB2 in astrocytes promotes radial glial progenitor indentity. Genes Dev. 21(24):3258-71.

Weimer, JM, Benedict, JW , Elshatory, YM, Short, DW, Ramirez-Montealegre, D, Ryan, DA, Alexander, NA, Federoff, HJ, Cooper, JD, and Pearce, DA (2007) Alterations in striatal dopamine catabolism precedes loss of substantia nigra neurons in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis. Brain Res., 1162:98-112.

Kovács, AD, Weimer, JM, and Pearce, DA (2006) Selectively increased sensitivity of cerebellar granule cells to AMPA receptor-mediated excitotoxicity in a mouse model of Batten disease. Neurobio. of Disease, 22(3):575-585. Weimer, JM, Custer, AW, Alexander, NA, Benedict, JW, Federoff, HJ, Cooper, JD, and Pearce, DA (2006) Visual Deficits in a Mouse Model of Batten Disease Are the Result of Optic Nerve Degeneration and Loss of Dorsal Lateral Geniculate Thalamic Neurons. Neurobio. of Disease, 22(2):284-293.

Weimer, JM and Anton, ES (2006) Doubling up on microtubule stabilizers: Synergistic functions of doublecortin-like kinase and doublecortin in the developing cerebral cortex. Neuron, 49:3-4.

Kwon, JM, Rothberg, PG, Weimer, JM, Mink, JW and Pearce, DA (2005) Novel Cln3 mutation predicted to cause complete loss of function does not result in a more severe phenotype than classical JNCL. Neurosci. Letters, 387(2):111-114.

Weimer, JM, Chattopadhyay, S, Custer, A, and Pearce, DA (2005) Elevation of Hook1 in a disease model of Batten disease does not affect a novel interaction between Ankyrin G and Hook1. BBRC, 330:1176-1181.

Phillips, S, Benedict, JW, Weimer, JM, and Pearce, DA (2005) CLN3, the Protein Associated with Batten Disease: Structure, Function and Localization. Journal of Neuroscience Research, 79:573-583.

Weimer, JM, Kriscenski-Perry, E, Elshatory, Y, and Pearce, DA (2002) The Neuronal Ceroid Lipofuscinoses: Mutations in different proteins result in similar disease. NeuroMolecular Medicine, 1:111-124.

Yao, PJ, Weimer, JM, and Coleman, PD (2001) Immunohistochemistry study of clathrin associated protein AP-2 in Alzheimer’s disease. Neurobiology of Aging, 21:921-929.

Chow, N, Cox, C, Callahan, LM, Weimer, JM, Guo, L, and Coleman, PD (1998). Expression profiles of multiple genes in single neurons of Alzheimer's disease. Proceedings of National Academy of Science, 95(16):9620-9625.